多食症

多食症
多食症
類型	進食障礙、醫學徵象[]、症狀、營養、代謝和發育症狀[]
分類和外部資源
醫學專科	內分泌學, 精神病學
ICD-11	MG43.1
ICD-10	R63.2
ICD-9-CM	783.6
DiseasesDB	29453
MeSH	D006963
	[編輯此條目的維基數據]

多食症（英語：polyphagia或hyperphagia），也作食慾過旺，是指極度的飢餓或食慾的增加。^[1]

醫學

在醫學中，多食症是一個醫學徵象，暗示著極度的飢餓飢餓狀態，以及反常食用固體食物，可由糖尿病、克萊恩-萊文症候群^{[來源請求]}、基因紊亂的普瑞德威利症候群和巴德-畢德氏症候群引發。^[2]此外，破壞迷走神經的接收器也可造成多食症。^[3]

病因

多食症的成因有：^[4]

通常在糖尿病酮症酸中毒早期會出現多食症的症狀。^[5]然而，在胰島素越來越匱乏時、糖尿病酮症酸中毒進一步發展時，食慾就能控制住了。^[6]

參見

參考資料

^ Berthoud HR, Lenard NR, Shin AC. Food reward, hyperphagia, and obesity.. Am J Physiol Regul Integr Comp Physiol. 2011, 300 (6): R1266–77. PMC 3119156  . PMID 21411768. doi:10.1152/ajpregu.00028.2011.
^ OMIM::Prader-Willi （頁面存檔備份，存於網際網路檔案館）OMIM::Bardet-Biedl （頁面存檔備份，存於網際網路檔案館）
^ de Lartigue G, Ronveaux CC, Raybould HE. Deletion of leptin signaling in vagal afferent neurons results in hyperphagia and obesity. MOLECULAR METABOLISM. 2014, 3 (6): 595–607 [2017-09-01]. PMC 4142400  . PMID 25161883. doi:10.1016/j.molmet.2014.06.003. （原始內容存檔於2022-08-02）.
^ Updated by: Linda J. Vorvick, MD, Medical Director and Director of Didactic Curriculum, MEDEX Northwest Division of Physician Assistant Studies, Department of Family Medicine, UW Medicine, School of Medicine, University of Washington, Seattle, WA. Also reviewed David Zieve, MD, MHA, Isla Ogilvie, PhD, and the A.D.A.M. Editorial team. Appetite - increased. nih.gov. [2017-09-01]. （原始內容存檔於2016-07-05）.
^ Elliott RE, Jane JA, Wisoff JH. Surgical management of craniopharyngiomas in children: meta-analysis and comparison of transcranial and transsphenoidal approaches.. Neurosurgery. 2011, 69 (3): 630–43; discussion 643. PMID 21499159. doi:10.1227/NEU.0b013e31821a872d.
^ Masuzaki H, Tanaka T, Ebihara K, Hosoda K, Nakao K. Hypothalamic melanocortin signaling and leptin resistance--perspective of therapeutic application for obesity-diabetes syndrome.. Peptides. 2009, 30 (7): 1383–6 [2020-10-05]. PMID 19394382. doi:10.1016/j.peptides.2009.04.008. （原始內容存檔於2020-03-14）.

外部連結

Diseases and conditions associated with Polyphagia （頁面存檔備份，存於網際網路檔案館）

[pmid21411768-1] Berthoud HR, Lenard NR, Shin AC. Food reward, hyperphagia, and obesity.. Am J Physiol Regul Integr Comp Physiol. 2011, 300 (6): R1266–77. PMC 3119156  . PMID 21411768. doi:10.1152/ajpregu.00028.2011.

[2] OMIM::Prader-Willi （頁面存檔備份，存於網際網路檔案館）OMIM::Bardet-Biedl （頁面存檔備份，存於網際網路檔案館）

[pmid25161883-3] Lartigue G, Ronveaux CC, Raybould HE. Deletion of leptin signaling in vagal afferent neurons results in hyperphagia and obesity. MOLECULAR METABOLISM. 2014, 3 (6): 595–607 [2017-09-01]. PMC 4142400  . PMID 25161883. doi:10.1016/j.molmet.2014.06.003. （原始內容存檔於2022-08-02）.

[4] Updated by: Linda J. Vorvick, MD, Medical Director and Director of Didactic Curriculum, MEDEX Northwest Division of Physician Assistant Studies, Department of Family Medicine, UW Medicine, School of Medicine, University of Washington, Seattle, WA. Also reviewed David Zieve, MD, MHA, Isla Ogilvie, PhD, and the A.D.A.M. Editorial team. Appetite - increased. nih.gov. [2017-09-01]. （原始內容存檔於2016-07-05）.

[pmid21499159-5] Elliott RE, Jane JA, Wisoff JH. Surgical management of craniopharyngiomas in children: meta-analysis and comparison of transcranial and transsphenoidal approaches.. Neurosurgery. 2011, 69 (3): 630–43; discussion 643. PMID 21499159. doi:10.1227/NEU.0b013e31821a872d.

[pmid19394382-6] Masuzaki H, Tanaka T, Ebihara K, Hosoda K, Nakao K. Hypothalamic melanocortin signaling and leptin resistance--perspective of therapeutic application for obesity-diabetes syndrome.. Peptides. 2009, 30 (7): 1383–6 [2020-10-05]. PMID 19394382. doi:10.1016/j.peptides.2009.04.008. （原始內容存檔於2020-03-14）.

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